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Importantly, targeting cholesterol biosynthetic enzymes such as CYP51A1 can influence ferroptosis independently of metabolite levels, implying that these enzymes may have non-canonical, cholesterol-independent functions in ferroptotic regulation

Hypothalamic-pituitary-adrenal hypofunction in Myalgic Encephalomyelitis (ME)/Chronic Fatigue Syndrome (CFS) as a consequence of activated immune-inflammatory and oxidative and nitrosative pathways

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